early events of tobacco mosaic virus infection in nicotiana glutinosa L.
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early events of tobacco mosaic virus infection in nicotiana glutinosa L.

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Published by Veenman & Zonen in Wageningen .
Written in English

Book details:

Edition Notes

Includes bibliographical references.

StatementJeanne Dijkstra.
The Physical Object
Pagination83p. :
Number of Pages83
ID Numbers
Open LibraryOL20223631M

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Nicotiana glauca Graham and Nicotiana glutinosa L. plants were used. gene at early time points of infection Tobacco mosaic virus (TMV) caused serious loss in . M. Weintraub, H.W. RagetliAn electron microscope study of tobacco mosaic virus lesions in Nicotiana glutinosa Journal of Cell Biology, 23 (), pp. Google ScholarAuthor: András Takács, József Horváth, Richard Gáborjányi, Gabriella Kazinczi.   The N gene of tobacco (Nicotiana tabacum) is a typical resistance (R) gene engendering localization of tobacco mosaic virus (TMV) infection and the elicitation of a hypersensitive necrotic response. The consensus model for R gene-derived resistance is at the level of protein:protein interactions, in which proteins of the pathogen interact with already Cited by: Expression of the hypersensitive response (HSR) to tobacco mosaic virus (TMV) infection in Nicotiana tabacum L. cv. Xanthi-nc (genotype NN) is controlled by .

The recent appearance of the fourth edition of Bawden's book, Plant Viruses and Virus Diseases (12), supplies a convenient and comprehensive review of current knowledge and speculation in the field of plant virology. The present review will expand on one aspect of this broad field: the initia­ tion and early cellular events of plant virus infection, especially as applied to .   The initial reports by Francis O. Holmes in , working with TMV infection of Nicotiana glutinosa, that local necrotic lesions were a sign of plant virus infection rapidly opened up the prospects to determine virus titer, isolate viruses, dissect antiviral defenses, and most importantly to quantify viruses using bioassays (Holmes, ).Cited by: Sufficient sulfate supply has been linked to the development of sulfur induced resistance or sulfur enhanced defense (SIR/SED) in plants. In this study we investigated the effects of sulfate (S) supply on the response of genetically resistant tobacco (Nicotiana tabacum cv. Samsun NN) to Tobacco mosaic virus (TMV).Plants grown with sufficient sulfate (+S plants) developed Cited by: Resistance to TMV, for example, is controlled by a dominant gene (N), derived from N. glutinosa, and based on hypersensitivity to virus infection (necrotic lesions on the infected leaves). Resistant cultivars with this type of resistance have been used commercially in the production of cigar tobacco on East Java.

  Plant nucleotide binding site–leucine-rich repeat (NBS-LRR) proteins are similar to the nucleotide binding oligomerization domain (NOD) protein family in their domain structure. It has been suggested that most NOD proteins rely on ligand-mediated oligomerization for function, and we have tested this possibility with the N protein of tobacco (Nicotiana tabacum).Cited by: Global seed trade contributed to development and improvement of world agriculture. An adverse effect of global seed trade is reflected in disease outbreaks in new growing areas, countries, and continents. Among the seed-borne viruses, Tobamovirus species are currently considered a peril for crop production around the world. The unique tobamoviral particles confer stability to the Cited by: 6.   In contrast, treatment of tobacco epidermal cells with Cucumber mosaic virus (CMV), a virus that infects tobacco but has a dissimilar structure, did not elicit a fast oxidative burst. Based on these results, the authors proposed that the induction of ROS by CP was specific to Tobamoviruses (Allan et al., ).Cited by: Infection of cucumber mosaic virus together with the associated satellite Y RNA causes yellowing of leaves in Nicotiana tabacum. This was correlated with the down-regulation of a gene involved in chlorophyll biosynthesis (ChlI) which is targeted by small RNAs derived from the satellite RNA (Shimura et al., ; Smith et al., ).Cited by: